IAI FigSearch
Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowReprints and Permissions
Right arrow Copyright Information
Right arrow Books from ASM Press
Right arrow MicrobeWorld
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Nair, S. P.
Right arrow Articles by Berger-Bächi, B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Nair, S. P.
Right arrow Articles by Berger-Bächi, B.

 Previous Article  |  Next Article 

Infection and Immunity, July 2003, p. 4167-4170, Vol. 71, No. 7
0019-9567/03/$08.00+0     DOI: 10.1128/IAI.71.7.4167-4170.2003
Copyright © 2003, American Society for Microbiology. All Rights Reserved.

The {sigma}B Regulon Influences Internalization of Staphylococcus aureus by Osteoblasts

Sean P. Nair,1* Markus Bischoff,2 Maria M. Senn,2 and Brigitte Berger-Bächi2

Cellular Microbiology Research Group, Eastman Dental Institute for Oral Health Care Sciences, University College London, London WC1X 8LD, United Kingdom,1 Institute for Medical Microbiology, University of Zürich, CH-8028 Zürich, Switzerland2

Received 29 July 2002/ Returned for modification 18 December 2002/ Accepted 21 April 2003

Individual strains of Staphylococcus aureus have different capacities to become internalized by osteoblasts. Here we report that the levels of {sigma}B expressed by S. aureus correlate with the capacity of this bacterium to be internalized by osteoblasts. However, {sigma}B is not essential for internalization and does not necessarily account for the differences in the capacities of strains to be internalized.

* Corresponding author. Mailing address: Cellular Microbiology Research Group, Eastman Dental Institute, University College London, 256 Gray's Inn Rd., London WC1X 8LD, United Kingdom. Phone: 44(0)20 79151118. Fax: 44(0)20 79151127. E-mail: snair{at}eastman.ucl.ac.uk.

Editor: V. J. DiRita

Infection and Immunity, July 2003, p. 4167-4170, Vol. 71, No. 7
0019-9567/03/$08.00+0     DOI: 10.1128/IAI.71.7.4167-4170.2003
Copyright © 2003, American Society for Microbiology. All Rights Reserved.



This article has been cited by other articles:



Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
J. Bacteriol. J. Virol. Eukaryot. Cell
Microbiol. Mol. Biol. Rev. Clin. Vaccine Immunol. All ASM Journals

Copyright © 2003 by the American Society for Microbiology. All rights reserved.