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Infection and Immunity, June 2004, p. 3634-3637, Vol. 72, No. 6
0019-9567/04/$08.00+0     DOI: 10.1128/IAI.72.6.3634-3637.2004
Copyright © 2004, American Society for Microbiology. All Rights Reserved.

Increased Susceptibility of ß7-Integrin-Deficient Neonatal Mice in the Early Stage of Cryptosporidium parvum Infection

Roselyne Mancassola,1 Sonia Lacroix-Lamandé,1 Mathieu Barrier,1 Muriel Naciri,1 Henri Salmon,2 and Fabrice Laurent1*

Laboratoire de Contrôle et Immunologie de Maladies à Protozoaires, Unité 86 BASE,1 Laboratoire Lymphocytes et Immunité des Muqueuses, Unité 918 PII, INRA de Tours, 37380 Nouzilly, France2

Received 28 November 2003/ Returned for modification 31 December 2003/ Accepted 16 February 2004

Numerous inflammatory cells are recruited in response to Cryptosporidium parvum infection. These cells include interferon gamma-producing T lymphocytes, which are of major importance for the resolution of infection. Here, we show that ß7 integrin is not essential for the control of infection in mice but that ß7-deficient neonatal mice are more susceptible during the early stages of infection.

* Corresponding author. Mailing address: Laboratoire de Contrôle et Immunologie de Maladies à Protozoaires, Unité 86 BASE, INRA de Tours, 37380 Nouzilly, France. Phone: (33) 02 47 42 77 45. Fax: (33) 02 47 42 77 74. E-mail: laurent{at}tours.inra.fr.

Editor: W. A. Petri, Jr.

Infection and Immunity, June 2004, p. 3634-3637, Vol. 72, No. 6
0019-9567/04/$08.00+0     DOI: 10.1128/IAI.72.6.3634-3637.2004
Copyright © 2004, American Society for Microbiology. All Rights Reserved.





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