IAI FigSearch
Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Other Versions of this Article:
IAI.01372-06v1
75/5/2634    most recent
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowReprints and Permissions
Right arrow Copyright Information
Right arrow Books from ASM Press
Right arrow MicrobeWorld
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Maadani, A.
Right arrow Articles by Garsin, D. A.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Maadani, A.
Right arrow Articles by Garsin, D. A.

 Previous Article  |  Next Article 

Infection and Immunity, May 2007, p. 2634-2637, Vol. 75, No. 5
0019-9567/07/$08.00+0     doi:10.1128/IAI.01372-06
Copyright © 2007, American Society for Microbiology. All Rights Reserved.

Enterococcus faecalis Mutations Affecting Virulence in the Caenorhabditis elegans Model Host{triangledown}

Arash Maadani,1 Kristina A. Fox,1 Elftherios Mylonakis,2 and Danielle A. Garsin1*

Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas,1 Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts2

Received 25 August 2006/ Returned for modification 21 September 2006/ Accepted 7 February 2007

Enterococcus faecalis transposon insertion mutants were screened for attenuated killing of the nematode model host Caenorhabditis elegans. The genes disrupted in the attenuated mutants encode a variety of factors including transcriptional regulators, transporters, and damage control and repair systems. Five of nine mutants tested were attenuated in a mouse peritonitis model.

* Corresponding author. Mailing address: Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, 6431 Fannin St./MSB 1.174, Houston, TX 77030. Phone: (713) 500-5454. Fax: (713) 500-5499. E-mail: Danielle.A.Garsin{at}uth.tmc.edu

{triangledown} Published ahead of print on 16 February 2007.

Editor: A. Camilli

Infection and Immunity, May 2007, p. 2634-2637, Vol. 75, No. 5
0019-9567/07/$08.00+0     doi:10.1128/IAI.01372-06
Copyright © 2007, American Society for Microbiology. All Rights Reserved.



This article has been cited by other articles:



Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
J. Bacteriol. J. Virol. Eukaryot. Cell
Microbiol. Mol. Biol. Rev. Clin. Vaccine Immunol. All ASM Journals

Copyright © 2007 by the American Society for Microbiology. All rights reserved.