IAI FigSearch
Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
Authors' Corrections for Miyamoto et al., Infect. Immun. 70 (8) 4261-4272.
This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowReprints and Permissions
Right arrow Copyright Information
Right arrow Books from ASM Press
Right arrow MicrobeWorld
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Miyamoto, K.
Right arrow Articles by McClane, B. A.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Miyamoto, K.
Right arrow Articles by McClane, B. A.

 Previous Article

Infection and Immunity, March 2003, p. 1611, Vol. 71, No. 3
0019-9567/03/$08.00+0     DOI: 10.1128/IAI.71.3.1611.2003
Copyright © 2003, American Society for Microbiology. All Rights Reserved.

AUTHOR'S CORRECTION

Organization of the Plasmid cpe Locus in Clostridium perfringens Type A Isolates

Kazuaki Miyamoto, Ganes Chakrabarti, Yosiharu Morino, and Bruce A. McClane

Department of Molecular Genetics and Biochemistry, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania 15261, and Department of Microbiology, Wakayama Medical College, Wakayama, Japan 641-00122

Volume 70, no. 8, p. 4261-4272, 2002. Repeated sequencing has determined that the IS1151 sequences associated with the plasmid cpe gene of some type A isolates are not defective; therefore, the following corrections should be made.

Page 4269, column 1, lines 6-15 from bottom: "Sequencing of the ~1.1-kb cpe-IS1151 PCR product amplified from isolate F4013 DNA confirmed the presence of sequences resembling IS1151 ~0.3 kbp downstream of the F4013 plasmid cpe gene (Fig. 1). The amplified F4013 sequences were determined to be ~90% homologous to IS1151 but appear to be defective, as they contain several nonsense mutations. In addition, this sequencing analysis revealed that the region of F4013 DNA containing IS1151-like sequences also has an overlapping ORF (in a different reading frame from IS1151) sharing partial amino acid homology with transposase 11" should read "Sequencing of the ~1.1-kb cpe-IS1151 PCR product amplified from isolate F4013 DNA confirmed the presence of sequences resembling IS1151 ~0.3 kbp downstream of the F4013 plasmid cpe gene (Fig. 1). The amplified F4013 sequences were determined to be ~90% homologous to IS1151."

Page 4270, column 2, line 2 from bottom: "(which are defective)" should be deleted.

Infection and Immunity, March 2003, p. 1611, Vol. 71, No. 3
0019-9567/03/$08.00+0     DOI: 10.1128/IAI.71.3.1611.2003
Copyright © 2003, American Society for Microbiology. All Rights Reserved.




This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowReprints and Permissions
Right arrow Copyright Information
Right arrow Books from ASM Press
Right arrow MicrobeWorld
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Miyamoto, K.
Right arrow Articles by McClane, B. A.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Miyamoto, K.
Right arrow Articles by McClane, B. A.

Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
J. Bacteriol. J. Virol. Eukaryot. Cell
Microbiol. Mol. Biol. Rev. Clin. Vaccine Immunol. All ASM Journals