Erratum for Zughaier et al., Infect. Immun. 73 (5) 2940-2950.
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Infection and Immunity, May 2006, p. 3077, Vol. 74, No. 5
0019-9567/06/$08.00+0     doi:10.1128/IAI.74.5.3077.2006

ERRATUM

Differential Induction of the Toll-Like Receptor 4-MyD88-Dependent and -Independent Signaling Pathways by Endotoxins

Susu M. Zughaier, Shanta M. Zimmer, Anup Datta, Russell W. Carlson, and David S. Stephens

Division of Infectious Diseases, Department of Medicine, and Department of Microbiology and Immunology, Emory University School of Medicine, and Laboratories of Microbial Pathogenesis, Department of Veterans Affairs Medical Center, Atlanta, and Complex Carbohydrate Research Center, University of Georgia, Athens, Georgia

Volume 73, no. 5, p. 2940-2950, 2005. Page 2940, column 2, lines 4 and 5: "(1, 23, 36)" should read "(1a, 23, 36)."

Page 2941, Table 1: Footnote a should read "The published lipid A structures of these endotoxins showed that meningococcal and V. cholerae lipid A is a symmetrical hexa-acylated structure (6a, 19a), that E. coli lipid A is an asymmetrical hexa-acylated structure (1b, 7a), that Klebsiella pneumoniae, Salmonella serovar Typhimurium, and Salmonella serovar Minnesota lipid A is an asymmetrical hepta-acylated structure (19, 21a, 39a), that Pseudomonas aeruginosa lipid A is an asymmetrical penta-acylated structure (8a), and that Serratia marcescens lipid A is an asymmetrical hexa-acylated structure (1)."

Page 2949: The following references should be added. Because of these additions, original reference 1 becomes reference 1a.

References

    1
  1. Adams, G. A., and P. P. Singh. 1970. The chemical constitution of lipid A from Serratia marcescens. Can. J. Biochem. 48:55-62.[Medline]
  2. 1a
  3. Akira, S., and K. Hoshino. 2003. Myeloid differentiation factor 88-dependent and -independent pathways in Toll-like receptor signaling. J. Infect. Dis. 187(Suppl. 2):S356-S363.
  4. 1b
  5. Backhed, F., S. Normark, E. K. Schweda, S. Oscarson, and A. Richter-Dahlfors. 2003. Structural requirements for TLR4-mediated LPS signalling: a biological role for LPS modifications. Microbes Infect. 5:1057-1063.[CrossRef][Medline]
  6. 6a
  7. Broady, K. W., E. T. Rietschel, and O. Luderitz. 1981. The chemical structure of the lipid A component of lipopolysaccharides from Vibrio cholerae. Eur. J. Biochem. 115:463-468.[Medline]
  8. 7a
  9. Chatterjee, S. N., and K. Chaudhuri. 2003. Lipopolysaccharides of Vibrio cholerae. I. Physical and chemical characterization. Biochim. Biophys. Acta 1639:65-79.[Medline]
  10. 8a
  11. Ernst, R. K., A. M. Hajjar, J. H. Tsai, S. M. Moskowitz, C. B. Wilson, and S. I. Miller. 2003. Pseudomonas aeruginosa lipid A diversity and its recognition by Toll-like receptor 4. J. Endotoxin Res. 9:395-400.[CrossRef][Medline]
  12. 19a
  13. Kahler, C. M., and D. S. Stephens. 1998. Genetic basis for biosynthesis, structure, and function of meningococcal lipooligosaccharide (endotoxin). Crit. Rev. Microbiol. 24:281-334.[Medline]
  14. 21a
  15. Kanegasaki, S., K. Tanamoto, T. Yasuda, J. Y. Homma, M. Matsuura, M. Nakatsuka, Y. Kumazawa, A. Yamamoto, T. Shiba, S. Kusumoto, et al. 1986. Structure-activity relationship of lipid A: comparison of biological activities of natural and synthetic lipid A's with different fatty acid compositions. J. Biochem. (Tokyo) 99:1203-1210.[Abstract/Free Full Text]
  16. 39a
  17. Silipo, A., R. Lanzetta, A. Amoresano, M. Parrilli, and A. Molinaro. 2002. Ammonium hydroxide hydrolysis: a valuable support in the MALDI-TOF mass spectrometry analysis of lipid A fatty acid distribution. J. Lipid Res. 43:2188-2195.[Abstract/Free Full Text]


Infection and Immunity, May 2006, p. 3077, Vol. 74, No. 5
0019-9567/06/$08.00+0     doi:10.1128/IAI.74.5.3077.2006





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