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Infection and Immunity, February 2004, p. 1159-1161, Vol. 72, No. 2
0019-9567/04/$08.00+0     DOI: 10.1128/IAI.72.2.1159-1161.2004
Copyright © 2004, American Society for Microbiology. All Rights Reserved.

Heat Shock Protein 60 Is the Major Antigen Which Stimulates Delayed-Type Hypersensitivity Reaction in the Macaque Model of Chlamydia trachomatis Salpingitis

Anne B. Lichtenwalner,1 Dorothy L. Patton,1* Wesley C. Van Voorhis,2 Yvonne T. Cosgrove Sweeney,1 and Cho-Chou Kuo3

Departments of Obstetrics and Gynecology,1 Medicine and Pathobiology,2 Pathobiology, University of Washington, Seattle, Washington3

Received 8 September 2003/ Returned for modification 8 October 2003/ Accepted 24 October 2003

Chlamydial delayed-type hypersensitivity antigens were analyzed by using the subcutaneous salpingeal autotransplant model of Macaca nemestrina infected with Chlamydia trachomatis serovar E. Heat shock protein 60 was the only antigen shown to induce delayed-type hypersensitivity among other antigens tested, including UV-inactivated organisms, recombinant major outer membrane protein, purified outer membrane proteins, and heat shock protein 10.

* Corresponding author. Mailing address: Dorothy L. Patton, Department of Obstetrics and Gynecology, University of Washington, Box 356460, Seattle, WA 98195-6460. Phone: (206) 543-5554. Fax: (206) 616-9479. E-mail: dpatton{at}u.washington.edu.

Editor: W. A. Petri, Jr.

Infection and Immunity, February 2004, p. 1159-1161, Vol. 72, No. 2
0019-9567/04/$08.00+0     DOI: 10.1128/IAI.72.2.1159-1161.2004
Copyright © 2004, American Society for Microbiology. All Rights Reserved.



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