Host Response and Inflammation

Role of Interferon-Stimulated Gene Factor 3γ and Beta Interferon in HLA Class I Enhancement in Synovial Fibroblasts upon Infection with Chlamydia trachomatis

Jürgen Rödel, Heinz Vogelsang, Katrin Prager, Matthias Hartmann, Karl-Hermann Schmidt, Eberhard Straube
DOI: 10.1128/IAI.70.11.6140-6146.2002

Article Information

DOI 
https://doi.org/10.1128/IAI.70.11.6140-6146.2002
PubMed 
12379691
Published By 
American Society for Microbiology Journals
History 
  • Received April 24, 2002
  • Returned for modification June 11, 2002
  • Accepted August 5, 2002
  • Published online November 1, 2002.
Copyright & Usage 
Copyright © 2002 American Society for Microbiology

Author Information

  1. Jürgen Rödel1*,
  2. Heinz Vogelsang2,
  3. Katrin Prager1,
  4. Matthias Hartmann1,
  5. Karl-Hermann Schmidt1 and
  6. Eberhard Straube1
  1. 1Institute of Medical Microbiology
  2. 2Institute of Clinical Chemistry, Friedrich Schiller University of Jena, D-07740 Jena, Germany
  • ↵* Corresponding author. Mailing address: Institute of Medical Microbiology, Friedrich Schiller University of Jena, Semmelweisstr. 4, D-07740 Jena, Germany. Phone: 49-3641-933105. Fax: 49-3641-933474. E-mail: juergen.roedel{at}med.uni-jena.de .

  • Editor: J. D. Clements

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KEYWORDS

Chlamydia Infections
Chlamydia trachomatis
DNA-Binding Proteins
Histocompatibility Antigens Class I
Interferon-beta
transcription factors

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