The Intracellular Cyclophilin PpiB Contributes to the Virulence of Staphylococcus aureus Independently of Its Peptidyl-Prolyl cis/trans Isomerase Activity | Infection and Immunity

DOI: 10.1128/IAI.00379-18

Article Information

DOI

https://doi.org/10.1128/IAI.00379-18

PubMed

Published By

American Society for Microbiology Journals

History

Received July 10, 2018
Returned for modification July 30, 2018
Accepted August 6, 2018
Published online October 25, 2018.

Copyright & Usage

Copyright © 2018 American Society for Microbiology. All Rights Reserved.

Editors and / or Reviewers

Nancy E. Freitag, Editor, University of Illinois at Chicago

Article Versions

Accepted Manuscript version (August 13, 2018).
You are viewing the most recent version of this article.

Author Information

Rebecca A. Keogh^a,
Rachel L. Zapf^a,
Richard E. Wiemels^a,
Marcus A. Wittekind^a and
Ronan K. Carroll^a

^aDepartment of Biological Sciences, Ohio University, Athens, Ohio, USA

Address correspondence to Ronan K. Carroll, carrolr3{at}ohio.edu.
Citation Keogh RA, Zapf RL, Wiemels RE, Wittekind MA, Carroll RK. 2018. The intracellular cyclophilin PpiB contributes to the virulence of Staphylococcus aureus independently of its peptidyl-prolyl cis/trans isomerase activity. Infect Immun 86:e00379-18. https://doi.org/10.1128/IAI.00379-18.

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KEYWORDS

Staphylococcus aureus

micrococcal nuclease

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