Bacterial Infections

The Intracellular Cyclophilin PpiB Contributes to the Virulence of Staphylococcus aureus Independently of Its Peptidyl-Prolyl cis/trans Isomerase Activity

Rebecca A. Keogh, Rachel L. Zapf, Richard E. Wiemels, Marcus A. Wittekind, Ronan K. Carroll
Nancy E. Freitag, Editor
DOI: 10.1128/IAI.00379-18

Article Information

DOI 
https://doi.org/10.1128/IAI.00379-18
PubMed 
30104214
Published By 
American Society for Microbiology Journals
History 
  • Received July 10, 2018
  • Returned for modification July 30, 2018
  • Accepted August 6, 2018
  • Published online October 25, 2018.
Copyright & Usage 
Copyright © 2018 American Society for Microbiology. All Rights Reserved.
Editors and / or Reviewers 
  • Nancy E. Freitag, Editor, University of Illinois at Chicago

Article Versions

Author Information

  1. Rebecca A. Keogha,
  2. Rachel L. Zapfa,
  3. Richard E. Wiemelsa,
  4. Marcus A. Wittekinda and
  5. Ronan K. Carrolla
  1. aDepartment of Biological Sciences, Ohio University, Athens, Ohio, USA
  • Address correspondence to Ronan K. Carroll, carrolr3{at}ohio.edu.

  • Citation Keogh RA, Zapf RL, Wiemels RE, Wittekind MA, Carroll RK. 2018. The intracellular cyclophilin PpiB contributes to the virulence of Staphylococcus aureus independently of its peptidyl-prolyl cis/trans isomerase activity. Infect Immun 86:e00379-18. https://doi.org/10.1128/IAI.00379-18.

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Aug 2018281040
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Nov 201813311317
Dec 20181993
Jan 2019391610
Feb 201940143
Mar 20194886
Apr 201923279
May 201922217
Jun 20193397
Jul 20191289
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KEYWORDS

PPIase
S. aureus
cyclophilin
nuclease
PpiB
Staphylococcus aureus
chaperone
micrococcal nuclease

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