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apoptosis

  • STAT3 Promotes Schistosome-Induced Liver Injury by Inflammation, Oxidative Stress, Proliferation, and Apoptosis Signal Pathway
    Fungal and Parasitic Infections
    STAT3 Promotes Schistosome-Induced Liver Injury by Inflammation, Oxidative Stress, Proliferation, and Apoptosis Signal Pathway

    Schistosomiasis is a parasitic helminth disease that can cause organ lesions leading to health damage. During a schistosome infection, schistosome eggs can flow into the liver along the portal vein.

    Jie Zhao, Xin Liu, Yao Chen, Lin-Shuang Zhang, Ya-Rong Zhang, Deng-Ren Ji, Shi-Meng Liu, Mo-Zhi Jia, Yong-Hong Zhu, Yong-Fen Qi, Feng-Min Lu, Yan-Rong Yu
  • <em>Chlamydia</em> Lipooligosaccharide Has Varied Direct and Indirect Roles in Evading both Innate and Adaptive Host Immune Responses
    Host Response and Inflammation
    Chlamydia Lipooligosaccharide Has Varied Direct and Indirect Roles in Evading both Innate and Adaptive Host Immune Responses

    Chlamydia bacteria are obligate intracellular pathogens which can cause a variety of disease in humans and other vertebrate animals. To successfully complete its life cycle, Chlamydia must evade both intracellular innate immune responses and adaptive cytotoxic T cell responses. Here, we report on the role of the chlamydial lipooligosaccharide (LOS) in evading the immune response. Chlamydia infection is known...

    Xisheng Wang, Daniel D. Rockey, Brian P. Dolan
  • Depletion of Alveolar Macrophages Increases Pulmonary Neutrophil Infiltration, Tissue Damage, and Sepsis in a Murine Model of <span class="named-content genus-species" id="named-content-1">Acinetobacter baumannii</span> Pneumonia
    Host Response and Inflammation
    Depletion of Alveolar Macrophages Increases Pulmonary Neutrophil Infiltration, Tissue Damage, and Sepsis in a Murine Model of Acinetobacter baumannii Pneumonia

    Acinetobacter baumannii has emerged as an important etiological agent of hospital-related infections, especially nosocomial pneumonia. The virulence factors of this bacterium and their interactions with the cells and molecules of the immune system just recently began to be extensively studied. Here, we investigated the impact of alveolar macrophages on...

    Hiu Ham Lee, Lilit Aslanyan, Arjun Vidyasagar, Melissa B. Brennan, Maxine S. Tauber, Maria A. Carrillo-Sepulveda, Michael R. Dores, Nathan W. Rigel, Luis R. Martinez
  • <span class="named-content genus-species" id="named-content-1">Coxiella burnetii</span> Requires Host Eukaryotic Initiation Factor 2α Activity for Efficient Intracellular Replication
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Coxiella burnetii Requires Host Eukaryotic Initiation Factor 2α Activity for Efficient Intracellular Replication

    Coxiella burnetii is the causative agent of human Q fever, eliciting symptoms that range from acute fever and fatigue to chronic fatal endocarditis. C. burnetii is a Gram-negative intracellular bacterium that replicates within an acidic lysosome-like parasitophorous vacuole (PV) in human macrophages...

    Katelynn R. Brann, Marissa S. Fullerton, Daniel E. Voth
  • Apoptosis Functions in Defense against Infection of Mammalian Cells with Environmental Chlamydiae
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Apoptosis Functions in Defense against Infection of Mammalian Cells with Environmental Chlamydiae

    Apoptotic cell death can be an efficient defense reaction of mammalian cells infected with obligate intracellular pathogens; the host cell dies and the pathogen cannot replicate. While this is well established for viruses, there is little experimental support for such a concept in bacterial infections. All Chlamydiales are obligate intracellular bacteria, and different species infect vastly different hosts....

    Dominik Brokatzky, Oliver Kretz, Georg Häcker
  • <span class="named-content genus-species" id="named-content-1">Stenotrophomonas maltophilia</span> Encodes a VirB/VirD4 Type IV Secretion System That Modulates Apoptosis in Human Cells and Promotes Competition against Heterologous Bacteria, Including <span class="named-content genus-species" id="named-content-2">Pseudomonas aeruginosa</span>
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Stenotrophomonas maltophilia Encodes a VirB/VirD4 Type IV Secretion System That Modulates Apoptosis in Human Cells and Promotes Competition against Heterologous Bacteria, Including Pseudomonas aeruginosa

    Stenotrophomonas maltophilia is an emerging opportunistic and nosocomial pathogen. S. maltophilia is also a risk factor for lung exacerbations in cystic fibrosis patients. S. maltophilia attaches to various mammalian cells, and we...

    Megan Y. Nas, Richard C. White, Ashley L. DuMont, Alberto E. Lopez, Nicholas P. Cianciotto
  • Dietary Polyunsaturated Fatty Acids Promote Neutrophil Accumulation in the Spleen by Altering Chemotaxis and Delaying Cell Death
    Host Response and Inflammation
    Dietary Polyunsaturated Fatty Acids Promote Neutrophil Accumulation in the Spleen by Altering Chemotaxis and Delaying Cell Death

    Neutrophils are the most abundant circulating leukocytes in humans and are essential for the defense against invading pathogens. Like many other cells of an organism, neutrophils can be highly influenced by the diet. We have previously described that mice fed a high-fat diet rich in polyunsaturated fatty acids (HFD-P) present a higher frequency of neutrophils in bone marrow than mice fed a high-fat diet rich in saturated fatty acids (...

    Sara L. Svahn, Saray Gutiérrez, Marcus A. Ulleryd, Intawat Nookaew, Veronica Osla, Fredrik Beckman, Staffan Nilsson, Anna Karlsson, John-Olov Jansson, Maria E. Johansson
  • Methamphetamine Impairs IgG1-Mediated Phagocytosis and Killing of <span class="named-content genus-species" id="named-content-1">Cryptococcus neoformans</span> by J774.16 Macrophage- and NR-9640 Microglia-Like Cells
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Methamphetamine Impairs IgG1-Mediated Phagocytosis and Killing of Cryptococcus neoformans by J774.16 Macrophage- and NR-9640 Microglia-Like Cells

    The prevalence of methamphetamine (METH) use is estimated at ∼35 million people worldwide, with over 10 million users in the United States. Chronic METH abuse and dependence predispose the users to participate in risky behaviors that may result in the acquisition of HIV and AIDS-related infections.

    Lilit Aslanyan, Hiu H. Lee, Vaibhav V. Ekhar, Raddy L. Ramos, Luis R. Martinez
  • Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Cell Cycle Arrest and Apoptosis Induced by Porphyromonas gingivalis Require Jun N-Terminal Protein Kinase- and p53-Mediated p38 Activation in Human Trophoblasts
    Hiroaki Inaba, Atsuo Amano, Richard J. Lamont, Yukitaka Murakami, Michiyo Matsumoto-Nakano
  • Open Access
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Diaphorina citri Nymphs Are Resistant to Morphological Changes Induced by “Candidatus Liberibacter asiaticus” in Midgut Epithelial Cells
    Marina Mann, Somayeh Fattah-Hosseini, El-Desouky Ammar, Richard Stange, EricaRose Warrick, Kasie Sturgeon, Robert Shatters, Michelle Heck

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