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Infection and Immunity
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Brucella melitensis

  • The Twin-Arginine Translocation System Is Important for Stress Resistance and Virulence of <span class="named-content genus-species" id="named-content-1">Brucella melitensis</span>
    Molecular Pathogenesis
    The Twin-Arginine Translocation System Is Important for Stress Resistance and Virulence of Brucella melitensis

    Brucella, the causative agent of brucellosis, is a stealthy intracellular pathogen that is highly pathogenic to a range of mammals, including humans. The twin-arginine translocation (Tat) pathway transports folded proteins across the cytoplasmic membrane and has been implicated in virulence in many bacterial pathogens. However, the roles of the Tat system and related substrates in Brucella remain unclear. We report...

    Xin Yan, Sen Hu, Yan Yang, Da Xu, Huoming Li, Wenxing Liu, Xijun He, Ganwu Li, Wentong Cai, Zhigao Bu
  • Intratracheal Inoculation with <span class="named-content genus-species" id="named-content-1">Brucella melitensis</span> in the Pregnant Guinea Pig Is an Improved Model for Reproductive Pathogenesis and Vaccine Studies
    Microbial Immunity and Vaccines | Spotlight
    Intratracheal Inoculation with Brucella melitensis in the Pregnant Guinea Pig Is an Improved Model for Reproductive Pathogenesis and Vaccine Studies

    Reproductive failure is the hallmark of brucellosis in animals. An uncommon but important complication in pregnant women who become acutely infected with Brucella melitensis is spontaneous pregnancy loss or vertical transmission to the fetus. Unfortunately, the mechanism behind reproductive failure is still obscure, partially due to the lack of a proper study model....

    Martha E. Hensel, Sankar P. Chaki, Lauren Stranahan, Anthony E. Gregory, Erin J. van Schaik, Daniel G. Garcia-Gonzalez, Omar Khalaf, James E. Samuel, Angela M. Arenas-Gamboa
  • Open Access
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Brucella Peptide Cross-Reactive Major Histocompatibility Complex Class I Presentation Activates SIINFEKL-Specific T Cell Receptor-Expressing T Cells
    Jerome S. Harms, Mike Khan, Cherisse Hall, Gary A. Splitter, E. Jane Homan, Robert D. Bremel, Judith A. Smith
  • Host Response and Inflammation
    Chronic Brucella Infection Induces Selective and Persistent Interferon Gamma-Dependent Alterations of Marginal Zone Macrophages in the Spleen
    Arnaud Machelart, Abir Khadrawi, Aurore Demars, Kevin Willemart, Carl De Trez, Jean-Jacques Letesson, Eric Muraille
  • Microbial Immunity and Vaccines
    Discordant Brucella melitensis Antigens Yield Cognate CD8+ T Cells In Vivo
    Marina A. Durward, Jerome Harms, Diogo M. Magnani, Linda Eskra, Gary A. Splitter
  • Microbial Immunity and Vaccines
    Nondividing but Metabolically Active Gamma-Irradiated Brucella melitensis Is Protective against Virulent B. melitensis Challenge in Mice
    D. M. Magnani, J. S. Harms, M. A. Durward, G. A. Splitter
  • Molecular Pathogenesis
    Establishment of Systemic Brucella melitensis Infection through the Digestive Tract Requires Urease, the Type IV Secretion System, and Lipopolysaccharide O Antigen
    Tatiane A. Paixão, Christelle M. Roux, Andreas B. den Hartigh, Sumathi Sankaran-Walters, Satya Dandekar, Renato L. Santos, Renée M. Tsolis
  • Microbial Immunity and Vaccines
    Immunization with a Single Dose of a Microencapsulated Brucella melitensis Mutant Enhances Protection against Wild-Type Challenge
    Angela M. Arenas-Gamboa, Thomas A. Ficht, Melissa M. Kahl-McDonagh, Allison C. Rice-Ficht
  • Molecular Pathogenesis
    Cytotoxicity in Macrophages Infected with Rough Brucella Mutants Is Type IV Secretion System Dependent
    Jianwu Pei, Qingmin Wu, Melissa Kahl-McDonagh, Thomas A. Ficht
  • Microbial Immunity and Vaccines
    Aerosol Infection of BALB/c Mice with Brucella melitensis and Brucella abortus and Protective Efficacy against Aerosol Challenge
    M. M. Kahl-McDonagh, A. M. Arenas-Gamboa, T. A. Ficht

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