Cervix Uteri
- Cellular Microbiology: Pathogen-Host Cell Molecular Interactions | SpotlightNeisseria gonorrhoeae Survival during Primary Human Cervical Epithelial Cell Infection Requires Nitric Oxide and Is Augmented by Progesterone
- Host Response and InflammationHost Chemokine and Cytokine Response in the Endocervix within the First Developmental Cycle of Chlamydia muridarum
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsNeisseria gonorrhoeae Infection Protects Human Endocervical Epithelial Cells from Apoptosis via Expression of Host Antiapoptotic Proteins
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsTrichomonas vaginalis Lipophosphoglycan Triggers a Selective Upregulation of Cytokines by Human Female Reproductive Tract Epithelial Cells
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsTrichomonas vaginalis-Induced Epithelial Monolayer Disruption and Human Immunodeficiency Virus Type 1 (HIV-1) Replication: Implications for the Sexual Transmission of HIV-1
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsGonococcal Phospholipase D Modulates the Expression and Function of Complement Receptor 3 in Primary Cervical Epithelial Cells
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsChlamydia trachomatis Disrupts N-Cadherin-Dependent Cell-Cell Junctions and Sequesters β-Catenin in Human Cervical Epithelial Cells