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Infection and Immunity
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interferon gamma

  • Modification of the Pulmonary MyD88 Inflammatory Response Underlies the Role of the <span class="named-content genus-species" id="named-content-1">Yersinia pestis</span> Pigmentation Locus in Primary Pneumonic Plague
    Host Response and Inflammation
    Modification of the Pulmonary MyD88 Inflammatory Response Underlies the Role of the Yersinia pestis Pigmentation Locus in Primary Pneumonic Plague

    Pneumonic plague, caused by Yersinia pestis, is a rapidly progressing bronchopneumonia involving focal bacterial growth, neutrophilic congestion, and alveolar necrosis. Within a short time after inhalation of Y. pestis, inflammatory cytokines are expressed via the Toll/interleukin-1 (IL-1) adaptor...

    Rachel M. Olson, Miqdad O. Dhariwala, William J. Mitchell, Jerod A. Skyberg, Deborah M. Anderson
  • Immediate Interferon Gamma Induction Determines Murine Host Compatibility Differences between <span class="named-content genus-species" id="named-content-1">Toxoplasma gondii</span> and <span class="named-content genus-species" id="named-content-2">Neospora caninum</span>
    Molecular Pathogenesis
    Immediate Interferon Gamma Induction Determines Murine Host Compatibility Differences between Toxoplasma gondii and Neospora caninum

    Rodents are critical for the transmission of Toxoplasma gondii to the definitive feline host via predation, and this relationship has been extensively studied as a model for immune responses to parasites. Neospora caninum is a closely related coccidian parasite of ruminants and canines but is not...

    Rachel S. Coombs, Matthew L. Blank, Elizabeth D. English, Yaw Adomako-Ankomah, Ifeanyi-Chukwu Samuel Urama, Andrew T. Martin, Felix Yarovinsky, Jon P. Boyle
  • Splenic Innate B1 B Cell Plasmablasts Produce Sustained Granulocyte-Macrophage Colony-Stimulating Factor and Interleukin-3 Cytokines during Murine Malaria Infections
    Host Response and Inflammation
    Splenic Innate B1 B Cell Plasmablasts Produce Sustained Granulocyte-Macrophage Colony-Stimulating Factor and Interleukin-3 Cytokines during Murine Malaria Infections

    The physiopathology of malaria, one of the most deadly human parasitic diseases worldwide, is complex, as it is a systemic disease involving multiple parasitic stages and hosts and leads to the activation of numerous immune cells and release of inflammatory mediators.

    Shu Shien Chin, Laurent Chorro, John Chan, Grégoire Lauvau
  • Open Access
    Neutrophils Dampen Adaptive Immunity in Brucellosis
    Host Response and Inflammation
    Neutrophils Dampen Adaptive Immunity in Brucellosis

    Brucella organisms are intracellular stealth pathogens of animals and humans. The bacteria overcome the assault of innate immunity at early stages of an infection.

    Ricardo Mora-Cartín, Cristina Gutiérrez-Jiménez, Alejandro Alfaro-Alarcón, Esteban Chaves-Olarte, Carlos Chacón-Díaz, Elías Barquero-Calvo, Edgardo Moreno
  • Molecular Pathogenesis
    Characterization of Chlamydial Rho and the Role of Rho-Mediated Transcriptional Polarity during Interferon Gamma-Mediated Tryptophan Limitation
    Scot P. Ouellette, Parker R. Messerli, Nicholas A. Wood, Heather Hajovsky
  • Host Response and Inflammation
    Involvement of Host Defense Mechanisms against Toxoplasma gondii Infection in Anhedonic and Despair-Like Behaviors in Mice
    Motamed Elsayed Mahmoud, Ragab Fereig, Yoshifumi Nishikawa
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