Leishmania major
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsDeletion of Interleukin-4 Receptor Alpha-Responsive Keratinocytes in BALB/c Mice Does Not Alter Susceptibility to Cutaneous Leishmaniasis
The skin microenvironment at the site of infection plays a role in the early events that determine protective T helper 1/type 1 immune responses during cutaneous leishmaniasis (CL) infection. During CL in nonhealing BALB/c mice, early interleukin-4 (IL-4) can instruct dendritic cells for protective Th1 immunity.
- Fungal and Parasitic InfectionsCharacterization of the Protein Tyrosine Phosphatase LmPRL-1 Secreted by Leishmania major via the Exosome Pathway
- Host Response and InflammationFine Mapping of Leishmania major Susceptibility Locus lmr2 and Evidence of a Role for Fli1 in Disease and Wound Healing
- Fungal and Parasitic InfectionsUpregulated Expression of B-Cell Antigen Family Tandem Repeat Proteins by Leishmania Amastigotes
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsComplement Receptor 3 Deficiency Influences Lesion Progression during Leishmania major Infection in BALB/c Mice
- Cellular Microbiology: Pathogen-Host Cell Molecular InteractionsLeishmania major Phosphoglycans Influence the Host Early Immune Response by Modulating Dendritic Cell Functions
- Fungal and Parasitic InfectionsLeishmania major Infection Activates NF-κB and Interferon Regulatory Factors 1 and 8 in Human Dendritic Cells
- Fungal and Parasitic InfectionsComparison of the Effects of Leishmania major or Leishmania donovani Infection on Macrophage Gene Expression