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neutrophils

  • Type 3 Immunity during <span class="named-content genus-species" id="named-content-1">Clostridioides difficile</span> Infection: Too Much of a Good Thing?
    Minireview
    Type 3 Immunity during Clostridioides difficile Infection: Too Much of a Good Thing?

    Clostridioides (formerly known as Clostridium) difficile is the leading cause of hospital-acquired gastrointestinal infections in the United States and one of three urgent health care threats identified by the Centers for Disease Control and Prevention. C. difficile disease is mediated by the production of toxins that disrupt the epithelial...

    Mahmoud M. Saleh, William A. Petri
  • The Msp Protein of <span class="named-content genus-species" id="named-content-1">Treponema denticola</span> Interrupts Activity of Phosphoinositide Processing in Neutrophils
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    The Msp Protein of Treponema denticola Interrupts Activity of Phosphoinositide Processing in Neutrophils

    Periodontal disease is a significant health burden, causing tooth loss and poor oral and overall systemic health. Dysbiosis of the oral biofilm and a dysfunctional immune response drive chronic inflammation, causing destruction of soft tissue and alveolar bone supporting the teeth. Treponema denticola, a spirochete abundant in the plaque biofilm of patients with...

    Megan M. Jones, Stephen T. Vanyo, Michelle B. Visser
  • Interleukin 17 Receptor E (IL-17RE) and IL-17C Mediate the Recruitment of Neutrophils during Acute <em>Streptococcus pneumoniae</em> Pneumonia
    Host Response and Inflammation
    Interleukin 17 Receptor E (IL-17RE) and IL-17C Mediate the Recruitment of Neutrophils during Acute Streptococcus pneumoniae Pneumonia

    Neutrophils contribute to lung injury in acute pneumococcal pneumonia. The interleukin 17 receptor E (IL-17RE) is the functional receptor for the epithelial-derived cytokine IL-17C, which is known to mediate innate immune functions. The aim of this study was to investigate the contribution of IL-17RE/IL-17C to pulmonary inflammation in a mouse model of acute Streptococcus...

    Patrick Steck, Felix Ritzmann, Anja Honecker, Giovanna Vella, Christian Herr, Rosmarie Gaupp, Markus Bischoff, Timotheus Speer, Thomas Tschernig, Robert Bals, Christoph Beisswenger
  • Dietary Polyunsaturated Fatty Acids Promote Neutrophil Accumulation in the Spleen by Altering Chemotaxis and Delaying Cell Death
    Host Response and Inflammation
    Dietary Polyunsaturated Fatty Acids Promote Neutrophil Accumulation in the Spleen by Altering Chemotaxis and Delaying Cell Death

    Neutrophils are the most abundant circulating leukocytes in humans and are essential for the defense against invading pathogens. Like many other cells of an organism, neutrophils can be highly influenced by the diet. We have previously described that mice fed a high-fat diet rich in polyunsaturated fatty acids (HFD-P) present a higher frequency of neutrophils in bone marrow than mice fed a high-fat diet rich in saturated fatty acids (...

    Sara L. Svahn, Saray Gutiérrez, Marcus A. Ulleryd, Intawat Nookaew, Veronica Osla, Fredrik Beckman, Staffan Nilsson, Anna Karlsson, John-Olov Jansson, Maria E. Johansson
  • Interleukin 1 Receptor-Like 1 (IL1RL1) Promotes Airway Bacterial and Viral Infection and Inflammation
    Host Response and Inflammation
    Interleukin 1 Receptor-Like 1 (IL1RL1) Promotes Airway Bacterial and Viral Infection and Inflammation

    Interleukin 1 receptor-like 1 (IL1RL1), also known as suppression of tumorigenicity 2 (ST2), is the receptor for interleukin 33 (IL-33) and has been increasingly studied in type 2 inflammation. An increase in airway IL-33/ST2 signaling in asthma has been associated with eosinophilic inflammation, but little is known about the role of ST2 in neutrophilic inflammation.

    Niccolette Schaunaman, Amelia Sanchez, Kris Genelyn Dimasuay, Nicole Pavelka, Mari Numata, Rafeul Alam, Richard J. Martin, Hong Wei Chu
  • Open Access
    Neutrophils Dampen Adaptive Immunity in Brucellosis
    Host Response and Inflammation
    Neutrophils Dampen Adaptive Immunity in Brucellosis

    Brucella organisms are intracellular stealth pathogens of animals and humans. The bacteria overcome the assault of innate immunity at early stages of an infection.

    Ricardo Mora-Cartín, Cristina Gutiérrez-Jiménez, Alejandro Alfaro-Alarcón, Esteban Chaves-Olarte, Carlos Chacón-Díaz, Elías Barquero-Calvo, Edgardo Moreno
  • Interleukin-10 Produced by Myeloid-Derived Suppressor Cells Provides Protection to Carbapenem-Resistant <em>Klebsiella pneumoniae</em> Sequence Type 258 by Enhancing Its Clearance in the Airways
    Host Response and Inflammation
    Interleukin-10 Produced by Myeloid-Derived Suppressor Cells Provides Protection to Carbapenem-Resistant Klebsiella pneumoniae Sequence Type 258 by Enhancing Its Clearance in the Airways

    Carbapenem-resistant Klebsiella pneumoniae sequence type 258 (CRKP-ST258) can cause chronic infections in lungs and airways, with repeated episodes of bacteremia. In this report we addressed whether the recruitment of myeloid cells producing the anti-inflammatory cytokine interleukin-10 (IL-10) modulates the clearance of CKRP-ST258 in the lungs and establishes...

    Hernán F. Peñaloza, Loreani P. Noguera, Danielle Ahn, Omar P. Vallejos, Raquel M. Castellanos, Yaneisi Vazquez, Francisco J. Salazar-Echegarai, Liliana González, Isidora Suazo, Catalina Pardo-Roa, Geraldyne A. Salazar, Alice Prince, Susan M. Bueno
  • Toll-like Receptor 2 Prevents Neutrophil-Driven Immunopathology during Infection with <span class="named-content genus-species" id="named-content-1">Mycobacterium tuberculosis</span> by Curtailing CXCL5 Production
    Host Response and Inflammation
    Toll-like Receptor 2 Prevents Neutrophil-Driven Immunopathology during Infection with Mycobacterium tuberculosis by Curtailing CXCL5 Production

    The W-Beijing strain family is globally distributed and is associated with multidrug-resistant tuberculosis (TB) and treatment failure. Therefore, in this study, we examined the contribution of Toll-like receptor 2 (TLR2) to host resistance against Mycobacterium tuberculosis HN878, a clinical isolate belonging to the W-Beijing family.

    Archana Gopalakrishnan, Jillian Dietzold, Sheetal Verma, Madhuri Bhagavathula, Padmini Salgame
  • Open Access
    Gamma Interferon and Interleukin-17A Differentially Influence the Response of Human Macrophages and Neutrophils to <span class="named-content genus-species" id="named-content-1">Pseudomonas aeruginosa</span> Infection
    Host Response and Inflammation
    Gamma Interferon and Interleukin-17A Differentially Influence the Response of Human Macrophages and Neutrophils to Pseudomonas aeruginosa Infection

    Macrophages are important orchestrators of inflammation during bacterial infection, acting as both effector cells and regulators of neutrophil recruitment and life span. Differently activated macrophage populations with distinct inflammatory and microbicidal potentials have been described.

    Sirina Muntaka, Yasir Almuhanna, Darryl Jackson, Sonali Singh, Afrakoma Afryie-Asante, Miguel Cámara, Luisa Martínez-Pomares
  • Heterogeneity of Ly6G<sup>+</sup> Ly6C<sup>+</sup> Myeloid-Derived Suppressor Cell Infiltrates during <span class="named-content genus-species" id="named-content-1">Staphylococcus aureus</span> Biofilm Infection
    Host Response and Inflammation
    Heterogeneity of Ly6G+ Ly6C+ Myeloid-Derived Suppressor Cell Infiltrates during Staphylococcus aureus Biofilm Infection

    Myeloid-derived suppressor cells (MDSCs) are a heterogeneous population of immature monocytes and granulocytes. While neutrophils (polymorphonuclear leukocytes [PMNs]) are classically identified as highly differentiated cells specialized for antimicrobial defense, our laboratory has reported minor contributions of PMNs to the immune response during Staphylococcus...

    Cortney E. Heim, Sean C. West, Hesham Ali, Tammy Kielian

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