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Streptococcus agalactiae

  • The Novel Streptococcal Transcriptional Regulator XtgS Negatively Regulates Bacterial Virulence and Directly Represses PseP Transcription
    Bacterial Infections
    The Novel Streptococcal Transcriptional Regulator XtgS Negatively Regulates Bacterial Virulence and Directly Represses PseP Transcription

    Streptococcus agalactiae (group B streptococcus [GBS]) has received continuous attention for its involvement in invasive infections and its broad host range. Transcriptional regulators have an important impact on bacterial adaptation to various environments. Research on transcriptional regulators will shed new light on GBS pathogenesis. In this study, we identified a...

    Guangjin Liu, Tingting Gao, Xiaojun Zhong, Jiale Ma, Yumin Zhang, Shiyu Zhang, Zongfu Wu, Zihao Pan, Yinchu Zhu, Huochun Yao, Yongjie Liu, Chengping Lu
  • Genome-Wide Assessment of <span class="named-content genus-species" id="named-content-1">Streptococcus agalactiae</span> Genes Required for Survival in Human Whole Blood and Plasma
    Bacterial Infections
    Genome-Wide Assessment of Streptococcus agalactiae Genes Required for Survival in Human Whole Blood and Plasma

    Streptococcus agalactiae (group B streptococcus, or GBS) is a common cause of bacteremia and sepsis in newborns, pregnant women, and immunocompromised patients. The molecular mechanisms used by GBS to survive and proliferate in blood are not well understood. Here, using a highly virulent GBS strain and transposon-directed insertion site sequencing (TraDIS), we...

    Luchang Zhu, Prasanti Yerramilli, Layne Pruitt, Matthew Ojeda Saavedra, Concepcion C. Cantu, Randall J. Olsen, Stephen B. Beres, Andrew S. Waller, James M. Musser
  • Modulation of Death and Inflammatory Signaling in Decidual Stromal Cells following Exposure to Group B <em>Streptococcus</em>
    Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
    Modulation of Death and Inflammatory Signaling in Decidual Stromal Cells following Exposure to Group B Streptococcus

    Group B Streptococcus (GBS) is an opportunistic bacterial pathogen that contributes to miscarriage, preterm birth, and serious neonatal infections. Studies have indicated that some multilocus sequence types (STs) of GBS are more likely to cause severe disease than others. We hypothesized that the ability of GBS to elicit varying host responses in maternal decidual tissue during pregnancy is an important factor regulating...

    Rebecca A. Flaherty, Maja Magel, David M. Aronoff, Jennifer A. Gaddy, Margaret G. Petroff, Shannon D. Manning
  • Host-Associated Microbial Communities
    A Vaginal Tract Signal Detected by the Group B Streptococcus SaeRS System Elicits Transcriptomic Changes and Enhances Murine Colonization
    Laura C. C. Cook, Hong Hu, Mark Maienschein-Cline, Michael J. Federle
  • Molecular Pathogenesis
    Coassociation between Group B Streptococcus and Candida albicans Promotes Interactions with Vaginal Epithelium
    Grace R. Pidwill, Sara Rego, Howard F. Jenkinson, Richard J. Lamont, Angela H. Nobbs
  • Bacterial Infections
    cas9 Enhances Bacterial Virulence by Repressing the regR Transcriptional Regulator in Streptococcus agalactiae
    Ke Ma, Qing Cao, Su Luo, Zhaofei Wang, Guangjin Liu, Chengping Lu, Yongjie Liu
  • Open Access
    Bacterial Infections
    The Streptococcus agalactiae Stringent Response Enhances Virulence and Persistence in Human Blood
    Thomas A. Hooven, Andrew J. Catomeris, Maryam Bonakdar, Luke J. Tallon, Ivette Santana-Cruz, Sandra Ott, Sean C. Daugherty, Hervé Tettelin, Adam J. Ratner
  • Host Response and Inflammation
    Interaction between Pathogenic Bacteria and Intrauterine Leukocytes Triggers Alternative Molecular Signaling Cascades Leading to Labor in Women
    Guadalupe Estrada-Gutierrez, Nardhy Gomez-Lopez, Veronica Zaga-Clavellina, Silvia Giono-Cerezo, Aurora Espejel-Nuñez, Marco Antonio Gonzalez-Jimenez, Salvador Espino y Sosa, David M. Olson, Felipe Vadillo-Ortega
  • Molecular Pathogenesis
    Human Serum Induces Streptococcal C5a Peptidase Expression
    Ute Gleich-Theurer, Simone Aymanns, Gregor Haas, Stefanie Mauerer, Julia Vogt, Barbara Spellerberg
  • Host Response and Inflammation
    Role of p38 and Early Growth Response Factor 1 in the Macrophage Response to Group B Streptococcus
    Sybille Kenzel, Sandra Santos-Sierra, Sachin D. Deshmukh, Inga Moeller, Bilge Ergin, Katherine A. Fitzgerald, Egil Lien, Shizuo Akira, Douglas T. Golenbock, Philipp Henneke

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